What Are The Effects Of Acid Rain?

What Are The Effects Of Acid Rain? Part I What is Acid Rain? Acid rain is a general term that refers to the deposition of acidic materials from the atmosphere on the surface of the earth. Therefore it is more appropriately called acid deposition. There are two types of acid deposition, wet deposition and dry deposition. Wet deposition, or acid precipitation, refers to acidic rain, snow, hail or sleet. It occurs when pollutants such as Sulphur and Nitrogen oxides (SOx, NOx) are present in the atmosphere and react with water vapor to form acidic solutions such as Sulphurous Acid (H2SO3), Sulphuric Acid (H2SO4), Nitrous Acid (HNO2) and Nitric Acid (HNO3) that fall to Earths surface as precipitation. Dry deposition refers to the deposition of particles and gases of Sulphur and Nitrogen oxides (SOx, NOx) on Earths surface. These particles and gases become acids when they react with water on the surface of the Earth. Natural precipitation has a pH of about 5.6. It is slightly acidic due to dissolved Carbon dioxide (CO2). I n comparison, acid deposition usually has a pH of about 4.3 5.0. As stated above, acid deposition is caused by pollutants in the atmosphere. The majority of these pollutants are released in to the atmosphere by the burning of fossil fuels. Fuels such as coal and oil contain Sulphur and when they are burned they elease it. When Sulphur is released into the atmosphere by combustion it reacts accordingly: S + O2 Æ’Â   SO2 Sulphur released by the burning of fuels such as oil and coal reacts with Oxygen in the air to yield Sulphur dioxide. 2SO2 + O2 2SO3 Sulphur dioxide reacts with the Oxygen in the air to yield Sulphur trioxide SO2 + H2O Æ’Â   H2SO3 SO3 + H2O Æ’Â   H2SO4 Sulphur dioxide and Sulphur trioxide react with water, either in the atmosphere or on the surface of the Earth, to yield Sulphurous Acid and Sulphuric Acid Combustion of fuels in cars releases another pollutant, Nitrogen gas. Once Nitrogen gas is released in to the atmosphere by combustion it reacts accordingly: N2 + O2 Æ’Â   2NO N2 + 2O2 Æ’Â   2NO2 Nitrogen gas reacts with Oxygen in the air to yield Nitric oxide and Nitrogen dioxide 2NO + O2 2NO2 Nitric oxide reacts with oxygen in the air to yield Nitrogen dioxide 2NO2 + H2O Æ’Â   HNO2 + HNO3 Nitrogen dioxide reacts with water, either in the atmosphere or on the surface of the Earth, to yield Nitrous Acid and Nitric Acid. Small amounts of these pollutants are also released into the atmosphere naturally. Decomposing vegetation releases gases that contribute to pollutants in the atmosphere. Lightning produces Nitric oxide (NO) and volcanic eruptions release Sulphur dioxide (SO2). Some atmospheric chemists such as Dr. Ronald Prinn believe that acid rain has been around for millions of years. Dr. Prinn believes that acid rain could be responsible for the extinction of dinosaurs. However, the first known observation of acid rain did not take place until the 19th century, around the time of the Industrial Revolution. It was Robert Angus Smith, an English scientist, who came up with the term acid rain in 1872 when he observed that acidic precipitation was damaging plants. Acid rain was not considered a major environmental concern until the 1970s when scientists observed the effects of acid rain on ecosystems. When it was made clear that acid rain has negative impacts on ecosystems and that it is not only a regional issue but rather an international one, governments began to get involved. In 1991, Canada and the United States signed the Canada-US Air Quality Agreement and committed to reduce SOx and NOx emissions. Part II How does Acid Rain affect our Society? Acid rain has an extremely negative impact on our society. The most devastating consequence of acid rain is the affect that it has on terrestial as well as aquatic ecosystems. In terrestrial ecosystems, acid rain destroys plants. Acid rain causes toxic metals such as aluminum, copper, lead, manganese and zinc to leach out of rocks and soils. Additionally, nutrients present in topsoil leach down to the subsoil. Plant growth is significantly decreased in soils lacking nutrients and rich in poisonous alumnium. The other toxic metals released by acidic soil kill earthworms and nitrogen-fixing bacteria, both of which are essential to plant growth. Acid rain can also wears away the protective coating of leaves. These consequences also have an effect on the agriculture industry. Many crops cannot grow in soils that are lacking nutrients and contain aluminum. Furthermore, acid rain destroys many food crops such as radishes, tomatoes and apples. The effects of acid rain extends to aquatic eco systems. The toxic substances that are leached out of rocks and soil can be washed in to lakes and rivers. Fish are easily poisoned by these toxic substances. The acidic environment that acid rain creates in lakes and rivers is also harmful to fish and other aquatic organisms. The acidity kills many organisms that are usually at the bottom of a complex aquatic food chain. The food chain then collapses and the populations of many organisms significantly decrease. Acid rain also interrupts fish reproduction as it can destroy fish eggs. There are many more negative effects of acid rain. Fresh water supply can be contaminated by the acidity as well as by the metals that are leached from rocks and soil due to acid rain. Acid rain can also corrode water transportation pipes, allowing metal to enter the water supply. Another consequence of acid rain is the effect that it has on a cityscape. Acid rain can significantly damage metal and stone structures. Finally, acid rain can have an affect on human health. Contaminated water can cause health concerns. The SOx and NOx in dry deposition can cause numerous respiratory diseases such as asthma, emphysema and bronchitis. Furthermore, Nitrogen oxides (NOx) can react with volatile organic compounds in the air near the surface of the Earth to yield Ozone, which can be very detrimental to the respiratory system. Part III What should we do about Acid Rain? There are two methods that can be taken when approaching the issue of acid rain. We can either ignore the issue or we can do something about it and attempt to control the problem. Both methods have their advantages and disadvantages. If we ignore acid rain, nothing will be done to control or stop it and we will continue to suffer from its consequences. The advantage of this method is that no resources have to be expended. Governments, industries and citizens do not have to invest any money in to attempting to solve the problem. Governments do not have to invest in new energy sources. Industries can go on using current techniques without having to invest in new strategies in order to assist in solving the problem. Citizens can also go on without trying to conserve resources in order to attempt to stop acid rain. However, the disadvantage of this method is that acid rain will remain a threat to terrestrial ecosystems, aquatic ecosystems, the agriculture industry, cityscapes, fresh wate r supplies and human health. If acid rain continues the effects could be devastating. The second method is to do something about the problem and attempt to control acid rain. In order to do so pollutant emissions must be reduced, alternative sources of energy must be found and used, resources must be conserved, polices must be put in place in order to protect air quality and lakes and rivers must be neutralized. The advantage to this method is that it has the potential to control or even stop acid rain. By doing so we can save many terrestrial and aquatic ecosystems, increase yield of crops, prevent damage to cityscapes, keep supplies of fresh water clean and eliminate any risk of human health being put at risk due to acid rain. However, the disadvantage to this method is that it is very expensive. It also requires governments, industries and citizens to take action. To reduce pollutant emissions, companies have to invest in new cleaner technology. Such technology includes Sulphur s crubbers in factories and catalytic converters in cars. These technologies are expensive and require a large investment from industries. Furthermore, if policies are put in place in order to maintain air quality, many companies will have to invest in new technology in order to meet these new regulations. In order to find and use new energy sources industries as well as governments will have to invest a large amount of money into research and implementation. Additionally, neutralizing lakes and rivers that have been damaged by acid rain with limestone will also require the government to spend a large amount of money. All of these costs will eventually be passed down to the consumer as well as the citizen. In order to control acid rain we must also conserve our natural resources. This will be very hard for a society that is driven by exploitation. Revenues of many companies might go down if consumers begin to conserve. Revenues also may go down if companies begin consider the environm ent before they consider profit. After examining the advantages and disadvantages of ignoring the situation and controlling the situation, I think that we must control it. It would be wrong to ignore acid rain and the advantages of controlling it really outweigh its disadvantages. Controlling and trying to eliminate acid rain will be expensive and require governments, industries and citizens to take action but it is the right thing to do. We must save our ecosystems, our crops, our cities, our water and our health from the detrimental effects of acid rain. Ebola Virus: History, Causes and Effects Ebola Virus: History, Causes and Effects K.G Shayani Upulika Ebola Virus Outbreak: The deadline and incurable Hemorrhagic fever. Contents (Jump to) 1.1 Introduction to Ebola virus 1.2 History of Ebola virus. 1.3 Characteristics of Ebola virus 1.4 Symptoms of Ebola virus 1.5 Diagnosis methods of Ebola virus. 1.6 Treatments for Ebola virus 1.7 Conclusion. References: 1.1 Introduction to Ebola virus Identification of natural reservoir for Ebola virus was not possible by researchers for decades but recently a scientist named Leroy presented most acceptable evidence that three species of fruit bats called Hypsignathus monstrosus, Epomops franqueti and Myonycteris torquata are wildlife reservoirs for Ebola virus (Biek et al, 2006). Ebola virus is associated with acute fatal hemorrhagic symptoms for human and non-human primates when they get escaped from their wildlife reservoir hosts, Ebola virus is consider as a filo virus and one of the most lethal viruses known the five species of Ebola virus have been identified as Ivory Coast Ebola virus, Sudan Ebola virus, Zaire virus, Reston Ebola virus Ebola and Bundibugyo Ebola virus (Yuan et al, 2012). In the present situation of Ebola virus has a 90% death rate and the main site of first immerging is the region of Guinea’s southeastern forest region and this spilled throughout the entire Africa and some regions of the world (Bausc h and Schwarz, 2014). 1.2 History of Ebola virus. Ebola virus was first identified in 1976 in Congo of central Africa, It initially named as Zaire as it first found in town of Yambuku and after Yambuku outbreak the virus was vanished, but Ebola reappeared again in 1989 (Draper, 2002).The biomedical scientists first discovered the family of virus filoviridea when Marburg virus first appeared in 1967 and at that time commercial laboratory workers with sever and unusual disease symptoms were admitted to hospital in Marburg, Germany (peters and Ledue, 1999). After the discovery of Ebola it has re-appeared cyclically; most of the Ebola outbreaks are restricted to rural regions in central Africa and have never participated more than 500 cases, first Ebola case were noticed in February in wild areas of south Guinea and it has grown so strongly and spread so rapidly in terms of affected people and earthly spread those are Guinea, large parts of Liberia and Sierra, Lennon are disease ridden (Klenk, 2014). The virus has persisted in Africa and many fatal outbreaks in human and non-human primates have been reported and the History of Ebola virus is still unclear when doing researches Ebola virus strains with complete glycoprotein genes, including Zaire, sudan.reston, Tai forest, and bundibugya were analyzed (Li and Chen, 2013). After finding the way of coming the Ebola virus to south Africa scientists have found that the virus have always been there in the region but people just never noticed, therefor it has recently introduced then the first report and phylogenetic investigations on the Guinea outbreak proposed that the Ebola virus found in Guinea is a noticeable strain from that observed in central Africa, Therefore Ebola virus is not a newcomer to the region, it has been circulating for some time in Guinea,and have become a sudden threat to human beings (Schwarz, and Bausch, 2014). Figure 1.1 Map of the three countries involved in the 2013-2014 outbreak of Ebola virus as of June 20 2014(Schwarz and Bausch, 2014). 1.3 Characteristics of Ebola virus The family filoviridae with order Mononegavirales contains two generas, as Marburg virus and Ebola virus (EBOV) (Kuhn et al., 2012). Ebola virus is again divided in to many species such as Zaire Ebola virus (ZEBOV) Sudan Ebola virus, Ta forest Ebola virus and Reston Ebola virus (REBOV) (Kuhn et al., 2012). VP40 is the major Ebola virus matrix protein and it plays a major role in virus assembly and budding. This virus have a diameter averagely 57 .58 nm and it differ slightly for VP40+VP35 at 63.64nm, VP40+VP35+NP at 66.75nm (Johnson, Bell and Harty, 2006).Ebola virus particle is uniform with uniform diameter of 80 nm but Ebola virus particles are greatly varying from their morphology including long up to about 14  µm, sometimes these particles are branched filaments or shorter filaments shaped like ‘6’, ‘u’ or a circle and the size of this viral genome considered as 19 kb (Cencciarelii et al, 2014). However this fever referred as a group of illness that ca used by viruses belongs to diverse range of families which includes Lassa fever, rift valley fever and so on and It may lead to overall vascular damage and usually accompanied by hemorrhage (Ratanshi et al, 2014). Filo virus outbreaks are specific because these viruses can intersperse by years or even decades without showing any disease activity and last new species of Ebola virus was found 14 years before in Ivory Coast (Towner et al., 2008). Ebola virus is characterized by secondary transmission and high case fatality and also by filamentous enveloped particles however treating Ebola virus was a challenged situation after infection because these virus consists recombinant human activated protein C(rhAPC) ,recombinant nematode anticoagulant protein c2 (rNCPc2), small interfering RNA(si RNA) ( Qui et al, 2014). 1.4 Symptoms of Ebola virus Ebola virus is an invasive pathogen that can cause a highly lethal hemorrhagic fever in both human and non-human beings this virus runs its course within 14 to 21 days and the infection initially present with non-specific symptoms like fever, myalgia and malaise as infection proceeds patients starts to show large bleeding and coagulation deformities including gastrointestinal bleeding, rash and a wide varieties of hematological irregularities (Sullivan, Yang and Nabel, 2014).The initial symptoms of Ebola virus are non-specific, including fever, rash and vomiting, The most generally experienced symptoms were non-bloody diarrhea (81%) and asthenia (77%) and Seven patient reported were observed with hemorrhagic symptoms, and out of seven six of patient died(Roddy et al, 2012).The incubation period after exposure to virus is typically 8 to 10 days but it can be as short as 2 days and extended to 21 days and the net death rate is high as 90%.Most people with Ebola die within 2 weeks of di sease onset (Turner, 2014). 1.5 Diagnosis methods of Ebola virus. Numbers of newer technologies were applied for the diagnosis of Ebola virus infection some of them are antigen detection assay, utilized extensively in the Reston outbreak and its investigation, these methods allowed a fast and unique recognition of Ebola virus in the blood of severly ill individuals even in rural areas also nonspecific detection of antibodies by the unintended fluorescent antibody test in humans during Reston investigation led to the adaptation of the enzyme (Kaslow, Stanberry and Due, 2014). Ebola virus disease can proved with various lab tests depending on the on the stage of the disease, within a few days of infection immunosobent assay, antigen detection tests, reverse transcription polymerase chain reaction (RT-PCR) assay, and virus location by cell culture is used (Plotkin, Oreanstein an Offit, 2008). after the patient get back to normal immunoglobulin M and Immunoglobulin G can be found, after death immunohistochemistry, PCR and isolation of the virus can be done the exact diagnosis can be done by polymerase chain reaction (PCR) and ELISA antigen testing furthermore clarifications on viral replication inside cells can be done through measurements of IgM antibodies ( Plotkin, Oreanstein an Offit, 2008). Diagnosis of Ebola comes in to play when symptoms and signs of fever, illness, petechial rash and disseminated bleeding are observed and most of the accurate diagnosis is done by using a polymerase chain reaction detection assay and can be confirmed by detection of viral replication in Vero cells (Plotkin, Orenstein and Offit, 2008). Ebola is mainly diagnosed from the symptoms and blood test here mainly looks for the presence of antibodies in the virus at earlier days detecting a infected patient was not easy and once recognized the patient must be isolated and relevant authorities should notified (Horobin, 2008). 1.6 Treatments for Ebola virus No effective treatments for Ebola virus at this time. Anti-viral medications currently available have no effect on Ebola virus. Care for patients with Ebola virus infection consists maintaining fluid and electrolyte equilibrium via oral or I.V. hydration, supporting oxygenation and BP, Adjustments of coagulation and treating comorbidities such as a concurrent bacterial infection (Turner, 2014). However without recommended vaccines or treatments Ebola virus management has been limited and barrier methods and prevention methods also become limited but a combination of monoclonal antibodies (ZMapp), optimized from two previous antibody cocktails, is able to rescue 100% of rhesus macaques when treatments is only initiated up to 5 days, High fever, abnormalities in blood count and vireamia were some evidences in animals before ZMapp invention after that advanced disease as indicated by increased liver enzymes, mucosal hemorrhage and generalized petechial could be reversed leading to a ful l recovery (Qiu et al, 2014). There are no licensed vaccines currently available against Ebola virus but many applicable vaccine platforms have been tested for their efficiency against the virus and this treatment includes exposed or lipid encapsulated DNA, particles like virus arrangements, Vesicular stomatitis virus, strain Indiana, Human parainfluenza virus 3 (HPIV-3) , vaccinia, Venezuelan equine encephalitis virus (VEEV) and replication-deficient human adenovirus serotype 5 (AdHu5) vectors(Richardson et al, 2011). Due to the absence of specific therapies, filo virus ward clinicians give infected patients with the supportive care regime consist of oral medication, oral fluid rehydration, nutritional additives and physiological aid (Roddy et al, 2012). 1.7 Conclusion. Ebola virus is considered as one of the most lethal virus found so far and it has extremely high mortality rate therefore according to the doctors and experts point of view anyone who has stayed in areas where Ebola outbreak have recently been reported should have knowledge about the symptoms of infection and seek medical advices at the first sign of illness. Many reasons are there for this highly lethal disease to be spread largely, the most important reason is hospitals lack medical equipment such as surgical gloves, masks and hypodermic syringes to inject medicines to patients and clean water and also the lack of high standard medical laboratories for the medical tests to done on the virus is also a major issue. The spread of knowledge about the Ebola virus is expected to solve another big part of the problem therefore prevention of a worldwide outbreak lies within the education of the virus, how Ebola victims can properly treated and by performing suitable action to isolate the v irus before it has spread and maintaining hygienic conditions as much as possible.   References: Biek, R., Walsh, P. D., Leroy, E.M. and Real, L. A. (2006) ‘Recent common ancestry of Ebola virus found in a bat reservoir’, Plos pathogens, 2(10) pp. 1-3. [Online] DOI: 10.1371/journal.ppat.0020090 (Accessed: 20 October 2014). Bausch, D. G. and Schwarz, L. (2014) ‘Outbreak of Ebola virus in Guinea: where ecology meets Economy’, PLOS Neglected tropical diseases, 8(7) pp. 1-5. [Online] DOI: 10.1371/journal.pntd.0003056 (Accessed: 20 October 2014). Cenciarelii, O., Pietropaoli, S., Frusteri, L., Malizia, A., Carestia, M., Amico, F. D., Sassolini,A., Giovanii, D. D., Tamburrini, A., Palombi, L.,Bellecci, C. and Gaudi, P. (2014) ‘Biological Emergency Management: The Case of Ebola 2014 and the air transportation involvement’, Microbial and Biochemical Technology, 6(5) pp. 1-7. [Online] DOI: 10.4172/1948-5948.1000152 (Accessed: 22 October 2014). Draper, A. S. (2002)’ Epidemics, deadly disease throughout the history Ebola’ .Google Books [Online]. Available at: http://books.google.lk/books?id=FohdK6o-8WICpg=PA1focus=viewportdq=history+of+ebolaoutput=html_text. (Accessed: 20 October 2014). Horobin, W. (2008) Diseases and Disorders. Google books [Online]. Available at: http://books.google.lk/books?id=-HRJOElZch8Cpg=PA303dq=diagnosis+of+ebolahl=ensa=Xei=FB4-VNrvJ5fpoASNrIGwAgved=0CEwQ6AEwCQ#v=onepageq=diagnosis of ebolaf=false. (Accessed: 25 October 2014). Johnson, R. F., Bell, P and Harty, R. N. (2006) ‘Effect of Ebola virus proteins GP,NP and VP35 on VP40 VLP morphology’, Springer, 9(11) pp.1-20.[Online] DOI: 10.1186/1743-422X-3-31 (Accessed: 22 October 2014). Khun, J.H., Becker, S., Ebihara, H., Geisbert, T. W. and Jahrling, P. B.(2012) ‘Virus taxonomy ninth report of the international committee on taxonomy of virus’, filoviridae , 8(8) pp. 665-671.[Online] .Available at: www.plospathogens.org (Accessed:28 September 2014). Klenk, H. D., (2014) ’Lessons to be learned from the Ebola virus outbreak in west Africa’, Emerging Microbes and Infections. 11(3) pp. 1-1. [Online]. DOI: 10.1038/emi.2014.68 (Accessed: 20 October 2014). Kaslow, R. A., Stanberry, L. R. and Due, J. W. L.(2014) Viral infection of human. Google books [Online]. Available at: http://books.google.lk/books?id=sxakBAAAQBAJpg=PA344dq=diagnosis+of+ebolahl=ensa=Xei=PRM9VPCVLpeMuASM6YKoDgved=0CDYQ6AEwAw#v=onepageq=diagnosis of ebolaf=false. (Accessed: 25 October 2014). Li, Y. H, and Chen, S. P.,(2013) ‘ Evolutionary history of Ebola virus’, Epidemiology and infection, 142(6). pp.1-3. [Online]. DOI: 10.1017/SO950268813002215 (Accessed: 20 October 2014). Peters, C. J. and Ledue, J. W. (1999) ‘An introduction to Ebola: the virus and the disease’, Ebola: The Virus and the Disease, 179(1) pp. 1-8. [Online] DOI: 10.1086/514322 (Accessed :20 October 2014). Plotkin, S. A., Orenstein, W. A. and Offit, P. A. (2008) Vaccines. Google Books [Online]. Available at: http://books.google.lk/books?id=hoigDQ6vdDQCpg=PA1061dq=diagnosis+of+ebolahl=ensa=Xei=PRM9VPCVLpeMuASM6YKoDgved=0CDEQ6AEwAg#v=onepageq=diagnosis of ebolaf=false. (Accessed: 25 October 2014). Qiu, X., Wong, G., Audet, J., Bello, A., Fernando, L., Alimonti, J. B., Bovendo, H. F., Wei, H., Aviles, J., Hiatt, E., Jhonson, A., Morton, J., Swope, K., Bohorova, O., Goodman, C., Kim, D., Pauly, M. H., Velasco, J., Pettitt, J., Olinger, G. G., Whaley, K., Xu, B., Strong, J. E., Zeitlin, L. and Kobinger, G. P.,(2014) ‘Reversion of advanced Ebola virus disease in nonhuman primates with ZMapp’, Nature, 1 (0), pp. 1-15.[Online] DOI: 10.1038/nature13777 (Accessed: 23 October 2014). Qiu, X., Wong, G., Audet, J., Bello, A., Fernando, L., Alimonti, B., Bovendo, H. F., Wei, H., Aviles, J., Hiatt, E., Johnson, A., Morton, J., Swope, K., Bohorov, O., Bohorova, N., Goodman, C., Kim, D., Pauly, M. H., Velasco, J., Pettitt, J., Olinger, G. G.,Whaley, K., Xu, B., Strong, J. E and Zeitlin, L. (2014) ‘Reversion of advanced Ebola virus disease in nonhuman primates with ZMapp’, Nature, 11 (7) pp. 1-4. [Online] DOI: 10.1038/nature13777 (Accessed: 26 October 2014). Ratanshi, R. S., Elbireer, A., Mayanja, F., Coutinho, A., Merry, C. (2014) ‘Ebola outbreak response; experience and development of screening tools for viral hemorrhagic fever (VHF) in HIV center of excellence near to VHF epicenters’, VHF screening during an Ebola outbreak in large Ugandan HIV clinic, 9 (7), pp. 1-8.[Online] Available at: www.plosone.org. (Accessed: 22 October 2014). Roddy, P., Howard, N., Kerkhove, M. D. V., Lutwama, J., Wamala, J., Yoti, Z., Colebunders, R., Palma, P. P., Sterk, E., Jeffs, B., Herp, M. V., Borchert, M. (2012) ‘Clinical Manifestations and Case Management of Ebola Hemorrhagic Fever Caused by a Newly Identified Virus Strain, Bundibugyo, Uganda, 2007–2008’, Ebola Clinical Manifestations and Case Management, 7 (12) pp. 1-12. [Online] Available at: www.plosone.org (Accessed: 24 October 2014). Richardson, J. S., Wong, G., Pillet, S., Schindle, S., Ennis, J., Turner, J., Strong, J. E., and Kobinger, G. P. (2011) ‘Evaluation of Different Strategies for Post-Exposure Treatment of Ebola Virus Infection in Rodents’, Journal of Bioterrorism and Biodefense, 11 (7) pp. 1-1. [Online] DOI: 10.4172/2157-2526.S1-007 (Accessed: 26 October 2014). Roddy, P., Howard, N., Kerkhove, M. D. V., Lutwama, J., Wamala, J., Yoti, Z., Colebunders, R., Palma, P. P., Sterk, E., Jefffs, B., Herp, M. V and Bochert, M. (2012) ‘Clinical Manifestations and Case Management of Ebola Haemorrhagic Fever Caused by a Newly Identified Virus Strain, Bundibugyo, Uganda, 2007–2008’, Ebola Clinical Manifestations and Case Management, 7(12) pp. 1-12. [Online] Available at: www.plosone.org (Accessed: 26 October 2014). Schwarz, L. and Bausch, D. G., (2014) ‘Outbreak of Ebola virus disease in Guinea:where ecology meets economy’, PLOS Neglected Tropical Diseases, 8(7) pp.1-5.[Online] Available at: www.plosntds.org. (Accessed: 20 October 2014). Sullivan, N., Young, Z. Y. and Nabel, G. J. (2014) ‘Ebola virus pathogenesis: Implications for vaccines and therapies’, Journal of virology, 88 (21) pp. 1-4. [Online] DOI: 10.1128/JVI.18.9733-9737.2003 (Accessed: 24 October 2014). Turner, C. (2014) ‘Ebola virus disease: An emerging threat’, Nursing, 44 (9) pp. 1-1. [Online] DOI: 10.1097/01.NURSE.0000453010.02525.ca (Accessed: 24 October 2014). Towner, J. S., Sealy, T. S., Khristova, M. L., Albarino, C. G., Reeder, S. A., Quan, P.L., Lipkin, W. L., Downing, R., Tappero, J. W., Okware, S., Lutwama, J., Bakamutumaho, B., Kaiwa, J., Comer, J. A., Rollin, P. E., Ksiazek, T. G., Nichol, S.T. (2008) ‘Newly discovered Ebola virus associated with hemorrhagic fever outbreak in uganda’, New species of Ebola virus in Uganda,4 (11), pp. 1-6. [Online] Available at: www.plospathogens.org (Accessed: 23 October 2014). Turner, C. (2014) ‘Ebola virus disease: An emerging threat’, Nursing, 44 (9) pp. 1-1. [Online] DOI: 10.1097/01.NURSE.0000453010.02525.ca (Accessed: 25 October 2014). Turner, C. (2014) ‘Ebola virus disease: An emerging threat’, Nursing, 44 (9) pp. 1-1. [Online] DOI: 10.1097/01.NURSE.0000453010.02525.ca (Accessed: 26 October 2014). Yuan, J., Zhang, Y., Li, J., Zhang, Y., Wang, L. F. and Shi, Z. (2012) ‘Serological evidence of Ebola virus infection in bats, China’, Virology Journal, 9(236) pp. 1-5. [Online] DOI: 10.1186/1743-422x-9-236 (Accessed: 20 October 2014). 1

The Geography of the Olympics

The London 2012 Olympics has had a massive effect on the world, in particular the UK. Geographically speaking the Olympic park is located in Stratford, East London. The Olympics is obviously a huge financial outlay, but it is not just for the 2 weeks of the games. Urban regeneration: The Stratford area was previously a under developed and unpleasant area of London. Due to the Olympics the ?4 billion urban regeneration of the 73 hectares of land within the M25 has been started, with Stratford at its heart. The new facilities will include over 100 shops, leisure facilities, hotels and schools. One area of Stratford is currently being developed to be the most significant business district since Canary Wharf, this is planned to provide space for up to 30’000 workers. Stratford’s residents have also benefitted from the athlete’s village, the poor standard of the previous housing has led to their demolition and residents will now be able to access the former athlete’s village as part of an affordable/social housing scheme. The inclusion of 11’000 new homes in the Olympic park area will provide what Boris Johnson dubbed â€Å"the most important urban regeneration project in the next 25 years. It was perhaps most needed in this area of London as it had one of the lowest GDP’s in Britain with unemployment peaking at 20% after the demise of what were once the world’s largest docks. The area was very undesirable and often dubbed stinky Stratford because of the slaughterhouses and noxious industries that operated there. A consequence of all this was the housing, before the Olympics it consisted of post-world war prefabs. (Sources: Gaurdian. co. uk/Live discussion- regeneration/ July 2012 & London2012. com/ Regeneration games/November 2011) Transport: With 204 countries competing in the 2012 and over 11 million tickets on sale, people from all around the globe will be travelling to see Olympic events. This influx of people needs significant public transport and a superb infrastructure in order to run smoothly. International arrivals/departures by plane relied on the continuous operation of major airports around the world and the transport links directly to major areas of London in particular, Stratford. This was facilitated by regular trains and buses running between the locations. Since the London 2012 games, Stratford train station has become the third largest station in the UK for the number of trains stopping there. The People: As well as including thousands of people from around the world the Olympics also managed to attract all age groups from young children to pensioners. This sets the Olympics apart from other events; the ability to include such a vast age range is incredibly hard it is event such as the Olympics that enable such a large demographic to come together. For the first time in Olympic history there was a female team member from each of the competing countries, this was a particular change for Saudi Arabia who let two women into the team this year in contrast to none in its Olympic history. The Finances: In the background of the photo it is clear to see the large steel structure. This was donated by Lakshmi Mittal, the owner of ArcelorMittal which is the world's leading integrated steel and mining company. The tower is named the ‘Orbit’ and is located in Orbit Circus to the East of the Olympic Stadium. Mr Mittal has long been a supporter of the Olympics, setting up a trust for aspiring Indian athletes after Indian won only one medal in the 2000 Olympics. Despite being of Indian origin he decided to fund the project in the UK, this clearly demonstrates a sense of globalisation as financial support came from philanthropists all over the world. The economic effects of the Olympics have also been demonstrated by many large corporations, such as Whitbread, the group including Premier Inn, 4 restaurant chains and Costa Coffee. They revealed that sales had pushed Whitbread to the top of the FTSE 100 risers, up 106p to ?22. 05. An average increase of around 5% across the group. Furthermore the construction of the Olympic sites has created 46’000 jobs, much of the workforce sourced locally to retain the financial benefits within the local area. Moreover the active encouragement of a healthy lifestyle generated by the Olympics has a significant impact upon the attitudes viewers have about sport and their health. Theoretically the long term effects of the Olympics could mean the government saves money in areas such as health care treatments associated with obesity and unhealthy living. This in turn would enable a higher proportion of taxes to be spent on further urban regeneration or other public interests. (Sources: Gaurdian. co. uk/Live discussion- regeneration/ July 2012 & Whitbread PLC/ Trading Update/ September 2012) The Economic benefits of the Olympics were hyped up to an un-believable level by some sources, figures circulated at around ?3. Billion from respected organizations such as Visa. This would have meant a 3. 5% rise in the UK’s overall economic output annually. It was also expected that between 2013-2015, all the UK regions would benefit financially totaling 2. 74 Billion in extra money spent by visitors. When broken down into sectors, the High street retailers were expected to gain the most totaling ?705 million, surprisingly the transport sector faired as the lowest e arner totaling just ?81 million. Therefore Visa came to the conclusion that the total ‘economic legacy’ would be ?5. 3 Billion by 2015. However, now the main Olympic Games have come to an end analysts, are broadcasting a contrasting opinions based on the financial figures from the games. The more realistic increase in the nation’s output for 2012 is likely to be around 1%, less than a third of the original predictions for the Olympics alone. This 1% would also include the Queen’s Diamond Jubilee which was estimated to have increase output by 0. 4% along with the 0. 1% of growth gained from ticket sales (doesn’t count as the Olympic effect) to a total of 0. %. So, even without the Olympic effect, output should increase by 0. 5% between the second and third quarters. For the economy to register even zero growth in 2012 as a whole output needs to rise by 1% this quarter and remain at that level in the final quarter. Several major think-tank’s and The Bank of England have cut the ir UK growth predictions to between 0% and -0. 7% after disappointing Olympic revenue. (Source: Visa Europe/Economic impact report/ July 2011 & Guardian. co. uk/Olympics-why the British economy isn’t a winner/August 2012)

Direct Democracy and Athens Essay - 755 Words

Athens was located on the Western Europe specifically it’s one of the city on Greece. It was the first birth of democracy and also it was the home of education, that’s included Philosophy, Artists and so many scientists were appeared in Athens. In this city there were so many government came out, but none of them didn’t satisfy what the Athenian people wanted, except one king was called Pericles. He was the first leader who proposed democracy and made Athens glorious by different directions, those were by their military, economy and by other things. The important thing why we learned about Athenian history is their astonishing change in the Western Europe . That means they were super smart in Architecture, artist and so many†¦show more content†¦In Athenian being a citizen means they have to fulfilled 4 things. Those were â€Å"vote in all elections, serve in office if elected, serve on juries and serve in the military during war†. This shows that being citizens means sacrificed themselves for Athens glorification. But the criteria was almost the same as today democracy, so they were smart and knowledgeable in all direction. During the Golden age the athens were in highly glorified. For instance, In the â€Å"Funeral Oration† Pericles talk about their apotheosis. He said â€Å" In short, I say that as a city we are the school of Hellas, while i doubt if the world can produce a man who, where he has only himself to depend upon, is equal to so many emergencies, and graced by so happy a versatility, as the Athenian†. ( Pericles funeral oration p.4) This quote means Athens are the model and the teacher of all Greece city state and also their man had different and god gave them different abilities for them, in other word their citizen had distinct. The second one is their building. That means they were highly in architecture. 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